Spinal manipulation techniques

There are many differing philosophies and concepts surrounding the practise of spinal manipulation and its effects on the pathologies which may exist in the spine. To satisfy all these philosophies an equal number of institutions has developed, teaching those wishing to learn. No matter what school presents its case or which philosophy is adhered to, all manipulative specialists claim to have a high success rate. They all use techniques which vary in nature, application and intent; they proclaim that their own methods are superior to those used by others; and yet, somehow they all obtain uniformly good results. Self-limitation of low back pain plays, of course, a significant role in this happy situation. Apart from this there are definite benefits which are obtained quickly by using manipulative techniques.

Throughout the years I have practised many forms of mobilisation and manipulation, including osteopathic and chiropractic techniques and those taught by Cyriax. I have come to believe that it is immaterial which manipulative school or philosophy one upholds for, no matter what particular manipulations are used, the results of technique appear to be the same. The general procedures of Cyriax are just as effective as the finely developed specific procedures of the osteopaths and chiropractors.

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It is now well documented that asymmetries in the facet joints and other developmental anomalies occur as regularly in the lumbar region as in the other areas of the spine, and are present in up to fifty-two percent of the population. Where palpable movement restrictions and departures from the typical exist, it is impossible to state that these are either the cause of the patient’s present symptoms or are likely to cause symptoms in the future. To conclude that palpable anomalies should be mobilised is no longer a tenable suggestion. One may well be dealing with perfectly ‘normal’ and asymptomatic asymmetrical lumbar articulations.

Diagnosis by palpation is relevent only when associated with the increase and decrease of mechanical deformation. Those who claim to ‘feel’ restrictions by palpation and simultaneously fail to reproduce the patient’s symptoms, are only fooling themselves and their patients. Enthusiasts searching for sacro-iliac pathology are frequently misled by this philosophy. The production of pain at a certain level by palpation and passive movement is not enough to justify treatment at that level. The pain produced must be the one that has forced consultation — not a new pain induced by the palpation.

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Various manipulative authorities maintain that mobilisation or manipulation of a hypermobile spinal segment should be avoided at all times. This is excellent advice when it applies to pathological hypermobilities as fractures and spondylolisthesis. However, a hypermobile joint in itself is considered by some to cause mechanical pain more readily than joints with a normal or decreased mobility. I must emphasise that a hypermobile joint becomes painful in the same manner as any other joint. When it is placed on full stretch for a long enough period or when the stretch is severe enough pain will be felt. It so happens that in the hypermobile segment a greater range of movement must be accomplished before full stretch is achieved. Thus, hypermobility is not in itself a painful state.


The basic misunderstanding that passive movements applied to a hypermobile joint are harmful, has led to the systematic development of techniques of specific manipulation which spare the hypermobile joints and affect only the adjacent hypomobile joints. It has also brought about unjustified condemnation of those who advocate general instead of specific manipulation. Many people have criticised and rejected the techniques of Cyriax which are considered to be unselective, non-specific and rather coarse in their application. However, at this time hundreds of Cyriax-trained doctors and physiotherapists successfully apply his methods in many parts of the world and it appears that no harm is caused by his approach. If general manipulation were damaging indeed, then by now there would be enough evidence to support this contention. I am sure that the antagonists of Cyriax would not have allowed any compromising situation to pass unnoticed.


Unfortunately, it appears that some manipulative schools attempt to enhance the quality and mystique of their skills by increasing the complexity of their techniques. They create the impression that to master the art and science of manipulation one must acquire very complex skills; these are possessed and taught only by an elite group, and if one wishes to enjoy the highest reputation in manipulation one must join this elite group; the only way to do this is to train at an exclusive establishment. This continuing trend is a threat to the scientific development of mechanical therapy.

The procedures which I have chosen for the treatment of low back pain are totally unsophisticated and non-specific. In general, the techniques affect many segments and localisation is not attempted. However, these procedures have an immediate cause and effect on the patient’s symptoms. The reasons put forth for their effectiveness may prove to be wrong in the future, but the effectiveness itself will not change: if patients are selected as suggested and the techniques are applied as suggested, the procedures will be as effective fifty years from now as they are today.


It is one of the main theses of this book that patients can be taught to manage, treat and control their own low back pain. In order to achieve this it is necessary to depart from the traditional methods of mechanical treatment, whereby the therapist does something to the patient to bring about change. In that case the patient attributes his recovery, rightly or wrongly, to what was done to him and for all future episodes of low back pain he commits himself to the care of the therapist. By avoiding the use of therapist-technique in the initial stages of treatment and substituting patient-technique, the patient will recognise that his recovery is clearly the result of his own efforts. Only few patients fail to assume responsibility for active participation in their treatment.


At present a great deal of research and academic discussion is taking place in attempts to establish the scientific validation of manipulation and explain its effects. The most obvious and most important effect of mobilisation and manipulation is the increase of range of movement at any joint to which the techniques are applied. This may be caused by a change in the position of an internal structure, or by such an alteration in an adjacent structure that a more normal function is possible than existed prior to the application of the technique. The increase in range of movement as obtained by manipulation and mobilisation can also be achieved by exercises when performed in a certain way. It is my belief that an exercise becomes a mobilisation when performed with a certain frequency and in such a way that a rhythmical passive stretch is created. And in a similar manner a passive mobilisation can become a manipulation. The suggestion put forth is that mobilisation and manipulation are nothing more than extended exercises, and an exercise can therefore become a mobilisation or manipulation. Therapy based on self-mobilisation and self-manipulation has arrived and it is possible to teach patients to practise mobilisation and manipulation of their own spine.


Provided there is adequate instruction and careful explanation regarding the aims of treatment, the self-treatment concept can be applied successfully to most low back pain patients — that is, to all the patients with the postural syndrome, nearly all with the dysfunction syndrome, and about seventy-five percent of the patients with the derangement syndrome. Thus, twenty-five to thirty percent of patients with low back pain will not recover on the exercise programme alone, and need additional techniques of either mobilisation or manipulation which must be applied by a specialist therapist. The only equipment required to treat patients is an adjustable treatment table, the height of which can be varied and which has an end section of about two feet that can be inclined up or down.

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After much investigation to determine the optimum number of movements necessary to effect stretching of shortened tissues and alteration in the position of the fluid nucleus, I have come to conclude that usually any significant change will occur within ten to fifteen repetitions of the procedure and no benefit will be obtained by exceeding this number. Therefore, exercises are performed in series of ten to fifteen excursions each. The number of times in the day that a series of exercises must be done varies according to the syndrome to be treated, the effects to be obtained, and the capabilities of the patient involved.

Unless stated otherwise, exercises will be performed with an almost continuous rhythm. On each contraction the maximum possible range must be maintained for a second or two. Each excursion must be followed by relaxation, and a brief pause of only a fraction of a second is required. Normally patients can complete between ten to fifteen excursions in one minute. Therefore no patient should use the excuse that he has not got enough time to do the exercises as instructed.


In assessing progress the evaluation of pain changes is vital. A patient can improve in several ways: the intensity of the pain may reduce; the frequency with which the pain occurs may decrease; or the site of the pain may alter. Centralisation of the pain indicates that the patient is improving, although in terms of intensity he may still feel one hundred percent of the pain originally complained of. In this case an explanation of the pain behaviour usually satisfies the patient regarding his progress.

If the site of pain has not changed on the patient’s next visit, I always enquire about the frequency and intensity of the pain in the following manner:

“If you had one hundred units of pain on your last visit, how many do you have today in terms of (a) frequency; and (b) intensity?”.

The procedures include patient-technique as well as therapist-technique. In order to facilitate quick reference the procedures are summarised below.

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  • Procedure 1 : lying prone
  • Procedure 2 : lying prone in extension
  • Procedure 3 : extension in lying
  • Procedure 4 : extension in lying with belt fixation
  • Procedure 5 : sustained extension
  • Procedure 6 : extension in standing
  • Procedure 7: extension mobilisation
  • Procedure 8 : extension manipulation
  • Procedure 9 : rotation mobilisation in extension
  • Procedure 10 : rotation manipulation in extension
  • Procedure 11 : sustained rotation/mobilisation in flexion
  • Procedure 12 : rotation manipulation in flexion
  • Procedure 13 : flexion in lying
  • Procedure 14 : flexion in standing
  • Procedure 15 : flexion in step standing
  • Procedure 16 : correction of lateral shift
  • Procedure 17 : self-correction of lateral shift
TREATMENT OF SIDE GLIDING DYSFUNCTION — CORRECTION OF SECONDARY LATERAL SHIFT Having observed thousands of lumbar spines it has become clear to me that asymmetry is the ‘norm’ and symmetry is almost atypical. Therefore, when examining dysfunction patients it is important to realise that many exhibit a minor scoliosis or lateral shift, the direction of which is sometimes extremely difficult to determine. With careful observation it can be seen that the top half of the patient’s body is not correctly related to the bottom half, and the patient has shifted laterally about the lumbar area. The anomalies include a number of lateral shifts now dysfunctional in character. These lateral shifts are referred to as secondary whereas those caused by derangement are primary. Fig. Recovery of loss of side gliding, leaching the procedure of self-correction of secondary lateral shift. As discussed previously, we must determine whether the lateral shift is relevant to the present symptoms or is merely a congenital or developmental anomaly. If side gliding produces pain the...
The Postural Syndrome DEFINITION I would define the postural syndrome as mechanical deformation of postural origin causing pain of a strictly intermittent nature, which appears when the soft tissues surrounding the lumbar segments are placed under prolonged stress. This occurs when a person performs activities which keep the lumbar spine in a relatively static position (as in vacuuming, gardening) or when they maintain end positions for any length of time (as in prolonged sitting). History Patients with postural pain are usually aged thirty or under. Frequently they have a sedentary occupation and in general they lack physical fitness. In addition to low back pain they often describe pains in the mid-thoracic and cervical areas. They state that the pain is produced by positions and not by movement, is intermittent and may sometimes disappear for two to three days at a time. It is often found that, when patients are more active at weekends — playing tennis and dancing — they have relatively little or no t...
Spine Health. PROCEDURE 5 — SUSTAINED EXTENSION To apply a sustained extension stress to the lumbar spine an adjustable couch, one end of which may be raised, is a necessary piece of equipment. The patient lies prone with his head at the adjustable end of the couch which is gradually raised, about one to two inches at the time over a five to ten minute period. Once the maximum possible degree of extension is reached, the position may be held for two to ten minutes, according to the patient’s tolerance. When lowering the patient the adjustable end of the couch should slowly be returned to the horizontal over a period of two to three minutes. This must not be done rapidly, for acute low back pain may result. Fig. Sustained extension. Effects: The procedure is predominantly used in the treatment of derangement. The effect is similar to that of the third procedure, but a time factor is added with the graduated increase and the sustained nature of the extension. In certain circumstances a sustained extension stress is preferable...
The Intervertebral Disc STRUCTURE In the lumbar spine the intervertebral discs are constructed similarly to those in other parts of the vertebral column. The disc has two distinct components: the annulus fibrosus forming the retaining wall for the nucleus pulposus. The annulus fibrosus is constructed of concentric layers of collagen fibres. Each layer lies at an angle to its neighbour and the whole forms a laminated band which holds the two adjacent vertebrae together and retains the nuclear gel. The annulus is attached firmly to the vertebral end plates above and below, except posteriorly where the peripheral attachment of the annulus is not so firm. Moreover, the posterior longitudinal ligament with which the posterior annulus blends is a relatively weak structure, whereas anteriorly the annulus blends intimately with the powerful anterior longitudinal ligament. The posterior part of the annulus is the weakest part: the anterior and lateral portions are approximately twice as thick as the posterior port...
Back pain. Predisposing and Precipitating Factors PREDISPOSING FACTORS Sitting posture There are three predisposing factors in the etiology of low back pain that overshadow most others. The first and most important factor is the sitting posture. A good sitting posture maintains the spinal curves normally present in the erect standing position. Postures which reduce or accentuate the normal curves enough to place the ligamentous structures under full stretch will eventually be productive of pain. Such postures are referred to as poor sitting postures. A poor sitting posture may produce back pain in itself without any additional other strains of living. We have all seen patients who entered an airliner, a car, or even a common lounge chair in a perfectly healthy and painfree state only to emerge hours later crippled with pain and unable to walk upright. Alternatively, a poor sitting posture will frequently enhance and always perpetuate the problems in patients suffering from low back pain. By far the great majority of patients comp...